This high-yield revision note on Peri-Implantitis vs Periodontitis: Key Differences covers everything examiners typically test under Periodontics — structured as quick-reference tables, clinical correlations, mnemonics and previous-year-style high-yield points. It's built for last-mile revision before NEET MDS, INI-CET MDS and INBDE, so you can scan it in minutes instead of re-reading a whole textbook chapter.
| Feature | Periodontitis | Peri-implantitis |
|---|---|---|
| Tissue involved | Cementum, periodontal ligament, and alveolar bone around a natural tooth | Peri-implant mucosa and bone around a dental implant - NO periodontal ligament or cementum, since there is direct bone-to-implant contact |
| Progression pattern | Often relatively slower; the PDL provides some buffering/defense | Tends to progress FASTER and in a non-linear, often accelerating pattern once initiated, since there is no PDL barrier to slow spread |
| Bone loss pattern | Vertical/horizontal, variable pattern | Often circumferential, "crater-like"/saucer-shaped bone loss around the implant |
| Probing reliability | PDL provides some tactile resistance/proprioceptive feedback | Peri-implant probing depths are less diagnostically reliable due to the absence of a true connective tissue attachment/biologic seal |
| Precursor (reversible) stage | Gingivitis | Peri-implant mucositis (analogous reversible precursor stage) |
| Treatment challenge | Generally better prognosis with SRP and regenerative options | Harder to treat - implant surface decontamination is technically challenging (threads harbor bacteria), with higher recurrence rates |
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