NEET MDS Shorts
34333
Oral PathologyPyostomatitis vegetans is a rare inflammatory condition of the oral mucosa that is strongly associated with inflammatory bowel disease (IBD), particularly ulcerative colitis. It is characterized by the presence of numerous small, yellow-white pustules on an erythematous base, which coalesce to form vegetative or papillary projections with a "snail track" appearance.
94051
Conservative DentistryExplanation: The outline form of a Class V cavity preparation is primarily determined by the extent of carious tissue. The goal is to remove all carious dentin while preserving as much healthy tooth structure as possible. The outline form must encompass the entire area affected by caries to ensure that the restoration is effective and durable. Other factors, such as the height of the gingival crest, type of restorative material, and tooth anatomy, may influence the preparation but are secondary to the extent of caries.
86847
INI CET
Bilirubin is the end product of heme catabolism.
[Image of the heme degradation pathway]
36542
Biochemistry
Uric acid is a purine derivative, increased by purine salvage reactions that convert purines, purine ribonucleosides, and purine deoxyribonucleoside to mononucleotides (incorrect answer 4). A defect in hypoxanthine-guanine phosphoribosyl transferase, one of the enzymes of purine salvage, is responsible for purine overproduction and subsequent hyperuricemia observed in Lesch-Nyhan syndrome.
Such salvage reactions require much less energy than de novo synthesis (incorrect answers 1, 2). The liver is the major site of purine nucleotide biosynthesis and provides excess purines for other tissues that cannot synthesize purines.
18575
Pharmacology
Sulfasalazine is absorbed well from GIT Sulfasalazine is poorly absorbed (10-20%) from the GIT .
86333
PedodonticsOcclusal correction is done after periodontal therapy primarily in cases of trauma from occlusion.
95677
Oral PathologyEnamel is most affected during formation due to Vitamin A deficiency.
97164
Pharmacology
Dimercaprol (British antilewisite; BAL) 1. Poisoning by As, Hg, Au, Bi, Ni, Sb: it is administered i.m., 5 mg/kg stat, followed by 2-3 mg/kg every 4 – 8 hours for 2 days, then once or twice a day for 10 days. It is partly oxidized and glucuronide conjugated, but mainly excreted as such in 4 – 6 hours. Earlier the treatment is instituted, the better it is. Because the dimercaprolmetal complex dissociates faster in acidic urine and the released metal can damage the kidney, urine is alkalinized during dimercaprol therapy. 2. As an adjuvant to Cal. Disod. Edentate in lead poisoning. 3. As an adjuvant to penicillamine in Cu poisoning and in Wilson’s disease – 300 mg/day i.m. for 10 days every second month. It is contraindicated in iron and cadmium poisoning, because the dimercaprol-Fe and dimercaprol-Cd complex is itself toxic.
91270
Oral SurgeryWhile the buccal nerve block is used to anesthetize the buccal mucosa and buccal gingival tissues of the maxillary molars, the actual technique does not typically produce any specific subjective symptoms in the patient other than the sensation of the needle. The patient may feel numbness if the anesthetic solution is inadvertently placed in the oral mucosa or if the block is done improperly.
82201
Dental Materials
Acidic dental cements typically have a pH less than 7.