Talk to us?

Periodontics - NEETMDS- courses
NEET MDS Lessons
General Medicine

  • GTR Membrane Placement:
    • Apical border: 3 – 4 mm below defect margin.
    • Lateral extension: 2 – 3 mm beyond defect.
  • Root Surface Biomodification: Citric acid (pH 1.0) for 2 – 5 min.
  • Enamel Matrix Derivative: Emdogain (from porcine teeth).
  • Cervical Enamel Projections: Seen in 8.6 – 28.6% of molars.
  • Smoking: Preventable risk factor; damages cilia via hydrogen cyanide.

Periodontal Fibers

Periodontal fibers play a crucial role in maintaining the integrity of the periodontal ligament and supporting the teeth within the alveolar bone. Understanding the different groups of periodontal fibers is essential for comprehending their functions in periodontal health and disease.

1. Gingivodental Group

  • Location:
    • Present on the facial, lingual, and interproximal surfaces of the teeth.
  • Attachment:
    • These fibers are embedded in the cementum just beneath the epithelium at the base of the gingival sulcus.
  • Function:
    • They help support the gingiva and maintain the position of the gingival margin.

2. Circular Group

  • Location:
    • These fibers course through the connective tissue of the marginal and interdental gingiva.
  • Attachment:
    • They encircle the tooth in a ring-like fashion.
  • Function:
    • The circular fibers help maintain the contour of the gingiva and provide support to the marginal gingiva.

3. Transseptal Group

  • Location:
    • Located interproximally, these fibers extend between the cementum of adjacent teeth.
  • Attachment:
    • They lie in the area between the epithelium at the base of the gingival sulcus and the crest of the interdental bone.
  • Function:
    • The transseptal fibers are primarily responsible for the post-retention relapse of orthodontically positioned teeth.
    • They are sometimes classified as principal fibers of the periodontal ligament.
    • Collectively, they form the interdental ligament of the arch, providing stability to the interproximal areas.

4. Semicircular Fibers

  • Location:
    • These fibers attach to the proximal surface of a tooth immediately below the cementoenamel junction (CEJ).
  • Attachment:
    • They go around the facial or lingual marginal gingiva of the tooth and attach to the other proximal surface of the same tooth.
  • Function:
    • Semicircular fibers help maintain the position of the tooth and support the gingival tissue around it.

5. Transgingival Fibers

  • Location:
    • These fibers attach to the proximal surface of one tooth and traverse the interdental space diagonally to attach to the proximal surface of the adjacent tooth.
  • Function:
    • Transgingival fibers provide support across the interdental space, helping to maintain the position of adjacent teeth and the integrity of the gingival tissue.

  • Drugs in GCF: Tetracycline, Metronidazole
  • Probes:
    • Conventional: Williams, Naber’s, CPITN
    • Pressure-sensitive: Foster Miller
    • Automated: Florida, Toronto
    • CPITN: 0.5 mm tip; markings at 3.5, 5.5, 8.5, 11.5 mm
  • Scaling & Curettage:
    • Blade angulation: 45 – 90° (scaling); >90° (curettage)
    • Curette sharpening: 100 – 110°
    • Chisel scaler: Push motion
    • Hoe scaler: Blade bent at 99°, used for ledges/rings

  • Plaque Biofilm:
    • Quorum sensing: bacterial communication
    • Co-aggregation: Gram-negative adherence
  • Microbial Shift (Health → Disease):
    • Facultative → Obligate anaerobes
    • Fermenting → Proteolytic
    • Cocci → Rods
    • Non-motile → Motile
  • Immunoglobulins:
    • Saliva: IgA
    • GCF: IgG
  • Interleukins & MMPs:
    • IL-1β: Most potent bone-destructive cytokine
    • MMP-8 & MMP-1: Collagenases
    • MMP-2 & MMP-9: Gelatinases
    • Activated by neutrophil proteases (e.g., cathepsin G)

Type Features
Type 1 DM Normal/underweight, Anti-GAD antibodies, HLA-DR3/DR4
Type 2 DM Overweight, Strong FHx, Hyperinsulinemia, Leptin role
MODY Autosomal dominant
LADA Type 1 in elderly
Brittle DM Wide sugar fluctuation, in children
Tropical DM Pancreatic calcification, abdominal pain
Acute Fulminant Viral infection
Latent Diabetes Unmasked in stress
Potential Diabetes FHx but normoglycemic

Classification by Extent

  • Generalized: >30% sites affected

Classification by Severity

  • Moderate: 3 – 4 mm bone loss
  • Severe: >5 mm bone loss

Aggressive Periodontitis

  • Rate: 3 – 4 times faster bone loss than chronic
  • Classic sign: Arc – shaped bone loss (distal 2nd premolar to mesial 2nd molar)
  • Associated: HLA A9 and B15 antigens
  • Receptor defect: Poorly functional monocyte FcγR2

Pain Characteristics

  • Periodontal pocket pain: "Deep in bone" feeling

Bone Loss Patterns

  • Radius of action: 1.5 – 2.5 mm (Page & Schroeder)
  • Reverse architecture: Facial/lingual plates lost, radicular bone preserved
  • TFO signs: Increased mobility (first clinical sign)

Epithelium Types

  • Parakeratinized: Retains pyknotic nuclei in stratum corneum
  • Orthokeratinized: No nuclei in stratum corneum
  • Keratinization order: Palate > Gingiva > Tongue > Cheek (most to least)

Keratin Distribution

  • K 1, 2, 10, 12: Orthokeratinized areas
  • K 6, 16: Highly proliferative areas
  • K 5, 14: Stratification specific
  • K 19: Parakeratinized areas

Structural Components

  • Stratum spinosum: Contains keratinosomes/Odland bodies (modified lysosomes)
  • Basal lamina: 300 – 400 Å thick, joins epithelium to connective tissue
    • Lamina lucida (rich in laminin)
    • Lamina densa (rich in collagen type IV)
  • Junctional epithelium: 0.25 – 1.35 mm long, formed by oral + reduced enamel epithelium

Clinical Probing

  • Normal probing depth: 2 – 3 mm (requires apical penetration)
  • Stippling: Orange peel appearance, restricted to attached gingiva
  • Loss of stippling: Early sign of gingivitis

Explore by Exams